Seroatlas · Human Serome Atlas

MAX

Protein max

Also known as: bHLHd4, bHLHd5, bHLHd6, bHLHd7, bHLHd8, MAX_HUMAN

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
P61244
Gene
MAX
Ensembl
ENSG00000125952
Chromosome
14
Canonical length
160 aa
Protein class
Cancer-related genes, Disease related genes, Human disease related genes, Predicted intracellular proteins, Transcription factors
Subcellular location
Nucleoplasm,Golgi apparatus

OverviewNCBI Gene

The protein encoded by this gene is a member of the basic helix-loop-helix leucine zipper (bHLHZ) family of transcription factors. It is able to form homodimers and heterodimers with other family members, which include Mad, Mxi1 and Myc. Myc is an oncoprotein implicated in cell proliferation, differentiation and apoptosis. The homodimers and heterodimers compete for a common DNA target site (the E box) and rearrangement among these dimer forms provides a complex system of transcriptional regulation. Mutations of this gene have been reported to be associated with hereditary pheochromocytoma. A pseudogene of this gene is located on the long arm of chromosome 7. Alternative splicing results in multiple transcript variants. [provided by RefSeq, Aug 2012]

Canonical amino-acid sequenceUniProt

160 residues, UniProt reviewed canonical sequence.

>P61244|MAX
     1  MSDNDDIEVE SDEEQPRFQS AADKRAHHNA LERKRRDHIK DSFHSLRDSV PSLQGEKASR
    61  AQILDKATEY IQYMRRKNHT HQQDIDDLKR QNALLEQQVR ALEKARSSAQ LQTNYPSSDN
   121  SLYTNAKGST ISAFDGGSDS SSESEPEEPQ SRKKLRMEAS

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against MAX can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Intracellular
Secreted
No
Transmembrane segments
0
Mean surface accessibility (rSASA)
0.54
Highest tissue expression
99 nTPM

Expression across tissuesHPA

Tissue

  • bone marrow: 99 nTPM
  • cerebellum: 72 nTPM
  • esophagus: 70 nTPM
  • vagina: 64 nTPM
  • spleen: 64 nTPM
  • fallopian tube: 62 nTPM

Single-cell type

  • platelets: 1,720 nCPM
  • esophageal apical cells: 764 nCPM
  • megakaryocytes: 383 nCPM
  • neutrophils: 293 nCPM
  • neutrophil progenitors: 262 nCPM
  • esophageal suprabasal cells: 233 nCPM

Immune cell

  • basophil: 670 nTPM
  • eosinophil: 542 nTPM
  • total PBMC: 314 nTPM
  • neutrophil: 307 nTPM
  • T-reg: 195 nTPM
  • naive CD4 T-cell: 152 nTPM

Brain region

  • cerebellum: 103 nTPM
  • medulla oblongata: 86 nTPM
  • white matter: 82 nTPM
  • pons: 80 nTPM
  • basal ganglia: 78 nTPM
  • thalamus: 78 nTPM

DiseaseUniProt · ClinVar · IEDB · PubMed

Four sources answering four different questions about MAX.

Disease | AllUniProt

Conditions MAX is implicated in, by any mechanism.

Disease | GeneticClinVar

54 pathogenic / likely-pathogenic of 638 ClinVar records.

Conditions with pathogenic or likely-pathogenic variants.

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
0.46
gnomAD pLI
0.83
gnomAD missense Z
1.81
DepMap mean gene effect
-0.84
DepMap dependency class
common

Cancer expressionTCGA

Across TCGA tumor cohorts, this protein is over-expressed in roughly 4% of surveyed tumor types (aggregate summary; per-cohort expression, alteration, and survival load in the interactive view).

OntologyGO

Biological processes

Molecular functions

Cellular components

Protein domainsUniProt · Pfam · InterPro

KeywordsUniProt

InteractionsUniProt · HPA

Protein binding partners of MAX in the human serome: UniProt-annotated complex subunits plus reported interactors. Each links to its own Seroatlas record.

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads MAX as an antibody target. Whether an autoantibody or antibody against MAX could matter depends on whether native MAX is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

MAX is annotated as predominantly intracellular. Intracellular proteins are common autoantibody markers, becoming visible to the immune system after cell injury or altered processing, but are usually markers of disease rather than direct drivers.

Annotation status

The present source text does not explicitly label MAX as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/MAX. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

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