Seroatlas · Human Serome Atlas

AKAP5

A-kinase anchor protein 5

Also known as: AKAP5_HUMAN, AKAP75, AKAP79

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
P24588
Gene
AKAP5
Ensembl
ENSG00000179841
Chromosome
14
Canonical length
427 aa
Protein class
Plasma proteins, Predicted intracellular proteins, Transporters
Subcellular location
Golgi apparatus,Plasma membrane,Cytosol

OverviewNCBI Gene

The A-kinase anchor proteins (AKAPs) are a group of structurally diverse proteins, which have the common function of binding to the regulatory subunit of protein kinase A (PKA) and confining the holoenzyme to discrete locations within the cell. This gene encodes a member of the AKAP family. The encoded protein binds to the RII-beta regulatory subunit of PKA, and also to protein kinase C and the phosphatase calcineurin. It is predominantly expressed in cerebral cortex and may anchor the PKA protein at postsynaptic densities (PSD) and be involved in the regulation of postsynaptic events. It is also expressed in T lymphocytes and may function to inhibit interleukin-2 transcription by disrupting calcineurin-dependent dephosphorylation of NFAT. [provided by RefSeq, Jul 2008]

Canonical amino-acid sequenceUniProt

427 residues, UniProt reviewed canonical sequence.

>P24588|AKAP5
     1  METTISEIHV ENKDEKRSAE GSPGAERQKE KASMLCFKRR KKAAKALKPK AGSEAADVAR
    61  KCPQEAGASD QPEPTRGAWA SLKRLVTRRK RSESSKQQKP LEGEMQPAIN AEDADLSKKK
   121  AKSRLKIPCI KFPRGPKRSN HSKIIEDSDC SIKVQEEAEI LDIQTQTPLN DQATKAKSTQ
   181  DLSEGISRKD GDEVCESNVS NSTTSGEKVI SVELGLDNGH SAIQTGTLIL EEIETIKEKQ
   241  DVQPQQASPL ETSETDHQQP VLSDVPPLPA IPDQQIVEEA SNSTLESAPN GKDYESTEIV
   301  AEETKPKDTE LSQESDFKEN GITEEKSKSE ESKRMEPIAI IITDTEISEF DVTKSKNVPK
   361  QFLISAENEQ VGVFANDNGF EDRTSEQYET LLIETASSLV KNAIQLSIEQ LVNEMASDDN
   421  KINNLLQ

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against AKAP5 can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Cell surface
Secreted
No
Transmembrane segments
0
Mean surface accessibility (rSASA)
0.69
Highest tissue expression
18 nTPM

Expression across tissuesHPA

Tissue

  • basal ganglia: 18 nTPM
  • cerebral cortex: 13 nTPM
  • amygdala: 5.5 nTPM
  • hippocampal formation: 4.6 nTPM
  • rectum: 3.6 nTPM
  • colon: 3.5 nTPM

Single-cell type

  • mast cells: 39 nCPM
  • goblet cells: 36 nCPM
  • nk-cells: 30 nCPM
  • brain inhibitory neurons: 29 nCPM
  • cardiomyocytes: 28 nCPM
  • brain excitatory neurons: 16 nCPM

Immune cell

  • basophil: 2.9 nTPM
  • gdT-cell: 0.3 nTPM
  • memory CD8 T-cell: 0.3 nTPM
  • naive B-cell: 0.2 nTPM
  • NK-cell: 0.2 nTPM
  • memory B-cell: 0.1 nTPM

Brain region

  • basal ganglia: 40 nTPM
  • cerebral cortex: 29 nTPM
  • amygdala: 23 nTPM
  • hippocampal formation: 22 nTPM
  • white matter: 12 nTPM
  • thalamus: 8.8 nTPM

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
0.52
gnomAD pLI
0.65
gnomAD missense Z
1.17
DepMap mean gene effect
0.01
DepMap dependency class
selective

Cancer expressionTCGA

Across TCGA tumor cohorts, this protein is over-expressed in roughly 4% of surveyed tumor types (aggregate summary; per-cohort expression, alteration, and survival load in the interactive view).

OntologyGO

Biological processes

Molecular functions

Cellular components

Protein domainsUniProt · Pfam · InterPro

KeywordsUniProt

InteractionsUniProt · HPA

Protein binding partners of AKAP5 in the human serome: UniProt-annotated complex subunits plus reported interactors. Each links to its own Seroatlas record.

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads AKAP5 as an antibody target. Whether an autoantibody or antibody against AKAP5 could matter depends on whether native AKAP5 is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

AKAP5 is annotated at the cell surface, where native AKAP5 is exposed to circulating antibodies and is a prime autoantibody target that could block, deplete, or overstimulate it.

Annotation status

The present source text does not explicitly label AKAP5 as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/AKAP5. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

Loading the interactive Seroatlas protein explorer...