Seroatlas · Human Serome Atlas

NRG4

Pro-neuregulin-4, membrane-bound isoform

Also known as: HRG4, NRG4_HUMAN

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
Q8WWG1
Gene
NRG4
Ensembl
ENSG00000169752
Chromosome
15
Canonical length
115 aa
Protein class
Predicted membrane proteins, Predicted secreted proteins
Secretome location
Secreted to digestive system

OverviewNCBI Gene

The neuregulins, including NRG4, activate type-1 growth factor receptors (see EGFR; MIM 131550) to initiating cell-to-cell signaling through tyrosine phosphorylation (Harari et al., 1999 [PubMed 10348342]).[supplied by OMIM, Mar 2008]

Canonical amino-acid sequenceUniProt

115 residues, UniProt reviewed canonical sequence.

>Q8WWG1|NRG4
     1  MPTDHEEPCG PSHKSFCLNG GLCYVIPTIP SPFCRCVENY TGARCEEVFL PGSSIQTKSN
    61  LFEAFVALAV LVTLIIGAFY FLCRKGHFQR ASSVQYDINL VETSSTSAHH SHEQH

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against NRG4 can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Cell surface
Secreted
No
Transmembrane segments
1
Mean surface accessibility (rSASA)
0.47
Highest tissue expression
8.9 nTPM

Expression across tissuesHPA

Tissue

  • cerebellum: 8.9 nTPM
  • pancreas: 5 nTPM
  • retina: 4 nTPM
  • hypothalamus: 2.3 nTPM
  • cerebral cortex: 1.9 nTPM
  • pituitary gland: 1.9 nTPM

Single-cell type

  • ependymal cells: 326 nCPM
  • pancreatic acinar cells: 222 nCPM
  • respiratory ciliated cells: 132 nCPM
  • myonuclei: 126 nCPM
  • foveolar cells: 114 nCPM
  • rod photoreceptor cells: 111 nCPM

Immune cell

  • basophil: 0 nTPM
  • classical monocyte: 0 nTPM
  • eosinophil: 0 nTPM
  • gdT-cell: 0 nTPM
  • intermediate monocyte: 0 nTPM
  • MAIT T-cell: 0 nTPM

Brain region

  • cerebellum: 8.8 nTPM
  • cerebral cortex: 2.7 nTPM
  • hypothalamus: 2.3 nTPM
  • spinal cord: 2.2 nTPM
  • pons: 2.1 nTPM
  • medulla oblongata: 2 nTPM

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
1.33
gnomAD pLI
0.03
gnomAD missense Z
0.41
DepMap mean gene effect
0.02
DepMap dependency class
none

Cancer expressionTCGA

Across TCGA tumor cohorts, this protein is over-expressed in roughly 4% of surveyed tumor types (aggregate summary; per-cohort expression, alteration, and survival load in the interactive view).

OntologyGO

Biological processes

Molecular functions

Cellular components

Protein domainsUniProt · Pfam · InterPro

KeywordsUniProt

InteractionsUniProt · HPA

Protein binding partners of NRG4 in the human serome: UniProt-annotated complex subunits plus reported interactors. Each links to its own Seroatlas record.

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads NRG4 as an antibody target. Whether an autoantibody or antibody against NRG4 could matter depends on whether native NRG4 is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

NRG4 is annotated at the cell surface, where native NRG4 is exposed to circulating antibodies and is a prime autoantibody target that could block, deplete, or overstimulate it.

Annotation status

The present source text does not explicitly label NRG4 as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/NRG4. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

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