Seroatlas · Human Serome Atlas

CIMAP1A

Ciliary microtubule associated protein 1A

Also known as: CMA1A_HUMAN, CT135, hSHIPPO, ODF3, SHIPPO1

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
Q96PU9
Gene
CIMAP1A
Ensembl
ENSG00000177947
Chromosome
11
Canonical length
254 aa
Protein class
Predicted intracellular proteins
Subcellular location
Mid piece,Principal piece,End piece

OverviewNCBI Gene

ODF3 is a component of sperm flagella outer dense fibers, which add stiffness, elastic recoil, and protection against shearing forces during sperm movement.[supplied by OMIM, Apr 2004]

Canonical amino-acid sequenceUniProt

254 residues, UniProt reviewed canonical sequence.

>Q96PU9|CIMAP1A
     1  MTEEVWMGTW RPHRPRGPIM ALYSSPGPKY LIPPTTGFMK HTPTKLRAPA YSFRGAPMLL
    61  AENCSPGPRY NVNPKILRTG KDLGPAYSIL GRYQTKTMLT PGPGDYFPEK STKYVFDSAP
   121  SHSISARTKA FRVDSTPGPA AYMLPMVMGP NTVGKASQPS FSIKGRSKLG GFSDDLHKTP
   181  GPAAYRQTDV RVTKFKAPQY TMAARVEPPG DKTLKPGPGA HSPEKVTLTK PCAPVVTFGI
   241  KHSDYMTPLL VDVE

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against CIMAP1A can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Intracellular
Secreted
No
Transmembrane segments
0
Mean surface accessibility (rSASA)
0.64
Highest tissue expression
9 nTPM

Expression across tissuesHPA

Tissue

  • testis: 9 nTPM
  • retina: 0.4 nTPM
  • adipose tissue: 0 nTPM
  • adrenal gland: 0 nTPM
  • amygdala: 0 nTPM
  • appendix: 0 nTPM

Single-cell type

  • early spermatids: 90 nCPM
  • late primary spermatocytes: 61 nCPM
  • late spermatids: 24 nCPM
  • thymic myoid cells: 1.4 nCPM
  • sertoli cells: 0.6 nCPM
  • cardiomyocytes: 0.5 nCPM

Immune cell

  • basophil: 0 nTPM
  • classical monocyte: 0 nTPM
  • eosinophil: 0 nTPM
  • gdT-cell: 0 nTPM
  • intermediate monocyte: 0 nTPM
  • MAIT T-cell: 0 nTPM

Brain region

  • cerebellum: 0.7 nTPM
  • cerebral cortex: 0.6 nTPM
  • medulla oblongata: 0.6 nTPM
  • white matter: 0.5 nTPM
  • basal ganglia: 0.4 nTPM
  • midbrain: 0.4 nTPM

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
0.77
gnomAD pLI
0.15
DepMap mean gene effect
0.05
DepMap dependency class
selective

Cancer expressionTCGA

Across TCGA tumor cohorts, this protein is over-expressed in roughly 4% of surveyed tumor types (aggregate summary; per-cohort expression, alteration, and survival load in the interactive view).

OntologyGO

Biological processes

Cellular components

Protein domainsUniProt · Pfam · InterPro

KeywordsUniProt

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads CIMAP1A as an antibody target. Whether an autoantibody or antibody against CIMAP1A could matter depends on whether native CIMAP1A is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

CIMAP1A is annotated as predominantly intracellular. Intracellular proteins are common autoantibody markers, becoming visible to the immune system after cell injury or altered processing, but are usually markers of disease rather than direct drivers.

Annotation status

The present source text does not explicitly label CIMAP1A as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/CIMAP1A. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

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