Seroatlas · Human Serome Atlas

ATXN7L3B

Ataxin-7-like protein 3B

Also known as: A7L3B_HUMAN, lnc-SCA7

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
Q96GX2
Gene
ATXN7L3B
Ensembl
ENSG00000253719
Chromosome
12
Canonical length
97 aa
Protein class
Disease related genes, Predicted intracellular proteins
Subcellular location
Nucleoplasm

OverviewNCBI Gene

Involved in regulation of gene expression. Located in cytoplasm. [provided by Alliance of Genome Resources, Jul 2025]

Canonical amino-acid sequenceUniProt

97 residues, UniProt reviewed canonical sequence.

>Q96GX2|ATXN7L3B
     1  MEEISLANLD TNKLEAIAQE IYVDLIEDSC LGFCFEVHRA VKCGYFYLEF AETGSVKDFG
    61  IQPVEDKGAC RLPLCSLPGE PGNGPDQQLQ RSPPEFQ

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against ATXN7L3B can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Intracellular
Secreted
No
Transmembrane segments
0
Mean surface accessibility (rSASA)
0.6
Highest tissue expression
43 nTPM

Expression across tissuesHPA

Tissue

  • retina: 43 nTPM
  • stomach: 36 nTPM
  • cerebral cortex: 35 nTPM
  • cerebellum: 34 nTPM
  • fallopian tube: 31 nTPM
  • thymus: 28 nTPM

Single-cell type

  • fallopian tube ciliated cells: 163 nCPM
  • respiratory ciliated cells: 137 nCPM
  • hematopoietic stem cells: 107 nCPM
  • megakaryocyte-erythroid progenitors: 94 nCPM
  • müller glia: 88 nCPM
  • endometrial ciliated cells: 87 nCPM

Immune cell

  • basophil: 17 nTPM
  • naive CD4 T-cell: 13 nTPM
  • eosinophil: 13 nTPM
  • non-classical monocyte: 12 nTPM
  • classical monocyte: 9.5 nTPM
  • total PBMC: 9.1 nTPM

Brain region

  • hypothalamus: 56 nTPM
  • pons: 50 nTPM
  • cerebral cortex: 48 nTPM
  • midbrain: 47 nTPM
  • basal ganglia: 47 nTPM
  • white matter: 46 nTPM

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
1.74
gnomAD pLI
0.15
gnomAD missense Z
0.6
DepMap mean gene effect
-0.15
DepMap dependency class
selective

Cancer expressionTCGA

Across TCGA tumor cohorts, this protein is over-expressed in roughly 6% of surveyed tumor types (aggregate summary; per-cohort expression, alteration, and survival load in the interactive view).

OntologyGO

Biological processes

Cellular components

Protein domainsUniProt · Pfam · InterPro

  • Ataxin-7-like protein 3B

KeywordsUniProt

InteractionsUniProt · HPA

Protein binding partners of ATXN7L3B in the human serome: UniProt-annotated complex subunits plus reported interactors. Each links to its own Seroatlas record.

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads ATXN7L3B as an antibody target. Whether an autoantibody or antibody against ATXN7L3B could matter depends on whether native ATXN7L3B is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

ATXN7L3B is annotated as predominantly intracellular. Intracellular proteins are common autoantibody markers, becoming visible to the immune system after cell injury or altered processing, but are usually markers of disease rather than direct drivers.

Annotation status

The present source text does not explicitly label ATXN7L3B as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/ATXN7L3B. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

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