Seroatlas · Human Serome Atlas

LENEP

Lens epithelial cell protein LEP503

Also known as: LENEP_HUMAN, LEP503

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
Q9Y5L5
Gene
LENEP
Ensembl
ENSG00000163352
Chromosome
1
Canonical length
61 aa
Protein class
Metabolic proteins, Predicted intracellular proteins

OverviewNCBI Gene

The ocular lens is a tissue of epithelial origin and devoid of blood vessels and nerves. Cells of the lens epithelium are responsible for the growth and maintenance of the lens through mitosis, protein synthesis, and active transport of ions and metabolites across the lens capsule. Lens epithelial protein is expressed exclusively in lens epithelial cells and may play a role in cell differentiation. [provided by RefSeq, Jul 2008]

Canonical amino-acid sequenceUniProt

61 residues, UniProt reviewed canonical sequence.

>Q9Y5L5|LENEP
     1  MQPRTQPLAQ TLPFFLGGAP RDTGLRVPVI KMGTGWEGFQ RTLKEVAYIL LCCWCIKELL
    61  D

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against LENEP can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Unknown
Secreted
No
Transmembrane segments
0
Mean surface accessibility (rSASA)
0.62
Highest tissue expression
0.3 nTPM

Expression across tissuesHPA

Tissue

  • salivary gland: 0.3 nTPM
  • kidney: 0.2 nTPM
  • skin: 0.2 nTPM
  • thyroid gland: 0.2 nTPM
  • cervix: 0.1 nTPM
  • endometrium: 0.1 nTPM

Single-cell type

  • müller glia: 1 nCPM
  • epididymal basal cells: 0.2 nCPM
  • melanocytes: 0.2 nCPM
  • ocular epithelial cells: 0.2 nCPM
  • rod photoreceptor cells: 0.2 nCPM
  • early primary spermatocytes: 0.1 nCPM

Immune cell

  • myeloid DC: 0.1 nTPM
  • naive CD8 T-cell: 0.1 nTPM
  • basophil: 0 nTPM
  • classical monocyte: 0 nTPM
  • eosinophil: 0 nTPM
  • gdT-cell: 0 nTPM

Brain region

  • cerebral cortex: 3.5 nTPM
  • cerebellum: 3.4 nTPM
  • white matter: 3.2 nTPM
  • thalamus: 2.8 nTPM
  • medulla oblongata: 2.6 nTPM
  • pons: 2.6 nTPM

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
1.93
gnomAD pLI
0
gnomAD missense Z
0.4
DepMap mean gene effect
-0.07
DepMap dependency class
selective

Cancer expressionTCGA

Across TCGA tumor cohorts, this protein is over-expressed in roughly 7% of surveyed tumor types (aggregate summary; per-cohort expression, alteration, and survival load in the interactive view).

OntologyGO

Biological processes

Molecular functions

Protein domainsUniProt · Pfam · InterPro

  • Lens epithelial cell protein LEP503
  • Lens epithelial cell protein LEP503

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads LENEP as an antibody target. Whether an autoantibody or antibody against LENEP could matter depends on whether native LENEP is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

LENEP is annotated as predominantly intracellular. Intracellular proteins are common autoantibody markers, becoming visible to the immune system after cell injury or altered processing, but are usually markers of disease rather than direct drivers.

Annotation status

The present source text does not explicitly label LENEP as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/LENEP. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

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