Seroatlas · Human Serome Atlas

ASAH2B

Putative inactive neutral ceramidase B

Also known as: ASA2B_HUMAN, ASAH2C, ASAH2L, bA449O16.3, bA98I6.3

Cross-references: UniProt · Ensembl · Human Protein Atlas · GeneCards · NCBI Gene

Protein identityUniProt · HPA

UniProt accession
P0C7U1
Gene
ASAH2B
Ensembl
ENSG00000204147
Chromosome
10
Canonical length
165 aa
Protein class
Predicted intracellular proteins
Subcellular location
Focal adhesion sites

OverviewNCBI Gene

Predicted to enable N-acylsphingosine amidohydrolase activity. Predicted to be involved in ceramide catabolic process. Predicted to be located in membrane. [provided by Alliance of Genome Resources, Jul 2025]

Canonical amino-acid sequenceUniProt

165 residues, UniProt reviewed canonical sequence.

>P0C7U1|ASAH2B
     1  MRQHRQFMDR THYLLTFSSS ETLLRLLLRI VDRAPKGRTF GDVLQPAKPE YRVGEVAEVI
    61  FVGANPKNSV QNQTHQTFLT VEKYEATSTS WQIVCNDASW ETRFYWHKGL LGLSNATVEW
   121  HIPDTAQPGI YRIRYFGHNR KQDILKPAVI LSFEGTSPAF EVVTI

LocalizationUniProt · AlphaFold · HPA

Whether an antibody against ASAH2B can act on the native protein depends on physical access: surface and secreted proteins are reachable by circulating antibodies, intracellular proteins usually are not.

Antibody reachability
Cell surface
Secreted
No
Transmembrane segments
0
Mean surface accessibility (rSASA)
0.37
Highest tissue expression
14 nTPM

Expression across tissuesHPA

Tissue

  • duodenum: 14 nTPM
  • small intestine: 9.3 nTPM
  • retina: 6.3 nTPM
  • liver: 5.7 nTPM
  • heart muscle: 4.9 nTPM
  • kidney: 4.1 nTPM

Single-cell type

  • cone photoreceptor cells: 35 nCPM
  • lactotrophs: 26 nCPM
  • myonuclei: 25 nCPM
  • podocytes: 21 nCPM
  • somatotrophs: 21 nCPM
  • rod photoreceptor cells: 20 nCPM

Immune cell

  • MAIT T-cell: 3.2 nTPM
  • naive CD4 T-cell: 2.6 nTPM
  • naive CD8 T-cell: 2.5 nTPM
  • T-reg: 2.4 nTPM
  • memory CD4 T-cell: 1.9 nTPM
  • plasmacytoid DC: 1.9 nTPM

Brain region

  • white matter: 11 nTPM
  • hypothalamus: 10 nTPM
  • cerebral cortex: 9.9 nTPM
  • medulla oblongata: 9.9 nTPM
  • midbrain: 9.6 nTPM
  • pons: 9.3 nTPM

Genetic constraint and essentialitygnomAD · DepMap

Does the body need this protein intact? Low LOEUF or a strong DepMap dependency means loss or blockade of the protein is likely to be felt.

gnomAD LOEUF (loss-of-function intolerance)
1.7
gnomAD pLI
0.16
gnomAD missense Z
0.76

OntologyGO

Biological processes

Molecular functions

Cellular components

Protein domainsUniProt · Pfam · InterPro

Antibody and autoantibody relevanceSeroatlas analysis

Seroatlas reads ASAH2B as an antibody target. Whether an autoantibody or antibody against ASAH2B could matter depends on whether native ASAH2B is physically reachable, whether the body needs it intact, and whether it acts in a disease-relevant tissue.

ASAH2B is annotated at the cell surface, where native ASAH2B is exposed to circulating antibodies and is a prime autoantibody target that could block, deplete, or overstimulate it.

Annotation status

The present source text does not explicitly label ASAH2B as an autoantigen. Seroatlas presents hypothesis context only and does not manufacture a known-serology claim.

Canonical record: https://seroatlas.com/gene/ASAH2B. Study-independent annotations aggregated from UniProt, Human Protein Atlas, PubMed, IEDB, Pfam, InterPro, Gene Ontology, AlphaFold, gnomAD, DepMap, ClinVar, TCGA. Catalog release seroatlas-reviewed-human-uniprot-20260313.

Seroatlas is the reference for exploring autoantibody and antibody serology at the human-protein level: the autoreactome and human serome, multiplex serology (HuProt, HuScan, VirScan, PhIP-Seq).

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